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Published Online: 6 July 2009

Chemical Modification with High Molecular Weight Polyethylene Glycol Reduces Transduction of Hepatocytes and Increases Efficacy of Intravenously Delivered Oncolytic Adenovirus

Publication: Human Gene Therapy
Volume 20, Issue Number 9

Abstract

Oncolytic adenoviruses are anticancer agents that replicate within tumors and spread to uninfected tumor cells, amplifying the anticancer effect of initial transduction. We tested whether coating the viral particle with polyethylene glycol (PEG) could reduce transduction of hepatocytes and hepatotoxicity after systemic (intravenous) administration of oncolytic adenovirus serotype 5 (Ad5). Conjugating Ad5 with high molecular weight 20-kDa PEG but not with 5-kDa PEG reduced hepatocyte transduction and hepatotoxicity after intravenous injection. PEGylation with 20-kDa PEG was as efficient at detargeting adenovirus from Kupffer cells and hepatocytes as virus predosing and warfarin. Bioluminescence imaging of virus distribution in two xenograft tumor models in nude mice demonstrated that PEGylation with 20-kDa PEG reduced liver infection 19- to 90-fold. Tumor transduction levels were similar for vectors PEGylated with 20-kDa PEG and unPEGylated vectors. Anticancer efficacy after a single intravenous injection was retained at the level of unmodified vector in large established prostate carcinoma xenografts, resulting in complete elimination of tumors in all animals and long-term tumor-free survival. Anticancer efficacy after a single intravenous injection was increased in large established hepatocellular carcinoma xenografts, resulting in significant prolongation of survival as compared with unmodified vector. The increase in efficacy was comparable to that obtained with predosing and warfarin pretreatment, significantly extending the median of survival. Shielding adenovirus with 20-kDa PEG may be a useful approach to improve the therapeutic window of oncolytic adenovirus after systemic delivery to primary and metastatic tumor sites.

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Published In

cover image Human Gene Therapy
Human Gene Therapy
Volume 20Issue Number 9September 2009
Pages: 975 - 988
PubMed: 19469693

History

Published in print: September 2009
Published online: 6 July 2009
Published ahead of production: 26 May 2009
Accepted: 22 May 2009
Received: 21 February 2009

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Authors

Affiliations

Konstantin Doronin
Division of Infectious Diseases, Department of Internal Medicine, Mayo Clinic, Rochester, MN 55902.
Elena V. Shashkova
Division of Infectious Diseases, Department of Internal Medicine, Mayo Clinic, Rochester, MN 55902.
Shannon M. May
Division of Infectious Diseases, Department of Internal Medicine, Mayo Clinic, Rochester, MN 55902.
Sean E. Hofherr
Division of Infectious Diseases, Department of Internal Medicine, Mayo Clinic, Rochester, MN 55902.
Michael A. Barry
Division of Infectious Diseases, Department of Internal Medicine, Mayo Clinic, Rochester, MN 55902.
Department of Immunology, Mayo Clinic, Rochester, MN 55902.
Department of Molecular Medicine, Mayo Clinic, Rochester, MN 55902.
Translational Immunovirology Program, Mayo Clinic, Rochester, MN 55902.
Cancer Center, Mayo Clinic, Rochester, MN 55902.

Notes

Address correspondence to:
Dr. Michael A. Barry
Mayo Clinic
200 First Street SW
Rochester, MN 55902
E-mail: [email protected]

Author Disclosure Statement

No competing financial interests exist.

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