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Published Online: 17 May 2011

Lung-Derived Mesenchymal Stromal Cell Post-Transplantation Survival, Persistence, Paracrine Expression, and Repair of Elastase-Injured Lung

Publication: Stem Cells and Development
Volume 20, Issue Number 10

Abstract

While multipotent mesenchymal stromal cells have been recently isolated from adult lung (L-MSCs), there is very limited data on their biological properties and therapeutic potential in vivo. How L-MSCs compare with bone marrow-derived MSCs (BM-MSCs) is also unclear. In this study, we characterized L-MSC phenotype, clonogenicity, and differentiation potential, and compared L-MSCs to BM-MSCs in vivo survival, retention, paracrine gene expression, and repair or elastase injury after transplantation. L-MSCs were highly clonogenic, frequently expressed aldehyde dehydrogenase activity, and differentiated into osteocytes, chondrocytes, adipocytes, myofibroblasts, and smooth muscle cells. After intravenous injection (2 h), L-MSCs showed greater survival than BM-MSCs; similarly, L-MSCs were significantly more resistant than BM-MSCs to anchorage independent culture (4 h) in vitro. Long after transplantation (4 or 32 days), a significantly higher number of CD45neg L-MSCs were retained than BM-MSCs. By flow cytometry, L-MSCs expressed more intercellular adhesion molecule-1 (ICAM-1), platelet derived growth factor receptor alpha (PDGFRα), and integrin α2 than BM-MSCs; these proteins were found to modulate endothelial adherence, directional migration, and migration across Matrigel in L-MSCs. Further, L-MSCs with low ICAM-1 showed poorer lung retention and higher phagocytosis in vivo. Compared with BM-MSCs, L-MSCs expressed higher levels of several transcripts (e.g., Ccl2, Cxcl2, Cxcl10, IL-6, IL-11, Hgf, and Igf2) in vitro, although gene expression in vivo was increased by L-MSCs and BM-MSCs equivalently. Accordingly, both L-MSCs and BM-MSCs reduced elastase injury to the same extent. This study demonstrates that tissue-specific L-MSCs possess mechanisms that enhance their lung retention after intravenous transplantation, and produce substantial healing of elastase injury comparable to BM-MSCs.

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cover image Stem Cells and Development
Stem Cells and Development
Volume 20Issue Number 10October 2011
Pages: 1779 - 1792
PubMed: 21585237

History

Published in print: October 2011
Published ahead of print: 6 July 2011
Published online: 17 May 2011
Accepted: 13 May 2011
Received: 6 March 2011

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Andrew M. Hoffman
Tufts University Cummings School of Veterinary Medicine, North Grafton, Massachusetts.
Julia A. Paxson
Tufts University Cummings School of Veterinary Medicine, North Grafton, Massachusetts.
Melissa R. Mazan
Tufts University Cummings School of Veterinary Medicine, North Grafton, Massachusetts.
Airiel M. Davis
Tufts University Cummings School of Veterinary Medicine, North Grafton, Massachusetts.
Shivraj Tyagi
Brigham and Womens Hospital, Harvard Medical School, Boston, Massachusetts.
Shankar Murthy
Brigham and Womens Hospital, Harvard Medical School, Boston, Massachusetts.
Edward P. Ingenito
Brigham and Womens Hospital, Harvard Medical School, Boston, Massachusetts.

Notes

Address correspondence to:Prof. Andrew M. HoffmanTufts University Cummings School of Veterinary MedicineBuilding 21, Suite 110200 Westboro RoadNorth Grafton, MA 01536E-mail: [email protected]

Author Disclosure Statement

The authors have no financial disclosure to declare.

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